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. 2022 Jul 20;13:330. doi: 10.1186/s13287-022-03010-y

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Blockade of exosomal generation of MSCs impaired the therapy for liver fibrosis. A Representative histological images of 2 weeks after PBS, MSC and MSC + GW4869 treatment. The top is HE staining, the middle is Sirius red staining and the bottom is Masson staining. Scale bars, 200 μm. B Western blot was used to measure the protein level of fibrotic Col1a1 and αSMA. C The mRNA levels of Col1a1 and αSMA were analyzed by RT-PCR assay