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. 2022 Jul 20;21:149. doi: 10.1186/s12943-022-01619-4

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — CircGPR137B inhibits in vitro cell growth. A RT-qPCR analysis of the expression levels of circGPR137B in different HCC cell lines. B RT-qPCR analysis of the overexpression efficiency of circGPR137B lentiviruses in HepG2 and Hep3B cell lines and the knockdown efficiency of sh-circGPR137B lentiviruses in SK-hep-1 cells. C MTT. Colony formation (D) and Transwell analysis (E) of the effects of circGPR137B overexpression or knockdown on cell proliferation, colony formation and cell invasion in HCC cell lines. Data are the means ± SEM of three experiments. *P < 0.05; **P < 0.01, ***P < 0.001