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. 2022 Jul 7;12:897703. doi: 10.3389/fonc.2022.897703

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Copyright © 2022 De Re, Rossetto, Rosignoli, Muraro, Racanelli, Tornesello, Zompicchiatti and Uzzau

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided tlammatory drugs ahe original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Caspase 8 modulates the crosstalk among cell survival, apoptosis, and necroptosis after engagement of tumor necrosis factor (TNF) death receptors. RIP,receptor-interacting protein; MLKL, Mixed Lineage Kinase Domain Like Pseudokinase; TLR, Toll-like receptor; IFNR, Interferon-gamma receptor; DAMPs, Damage-associated molecular patterns; PAMPs, Pathogen-associated molecular patterns; ROS, Reactive oxygen species; NF-kβ, Nuclear factor kappa-light-chain-enhancer of activated B cells; FAS-L, Fas Ligand; TNF-α, Tumor necrosis factor alfa.