Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2022 Jul 8;10:944813. doi: 10.3389/fped.2022.944813

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

Copyright © 2022 Tulzer, Huhta, Hochpoechler, Holzer, Karas, Kielmayer and Tulzer.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

PMC Copyright notice

Natural history of a fetus with critical aortic stenosis. The evolution of a hypoplastic left heart syndrome in a fetus with critical aortic stenosis is shown. The fetus was diagnosed at 24 + 1 weeks of gestation with a severely dilated and poorly contracting, but apex forming left ventricle (A). Five weeks later the fetus showed marked signs of left ventricular hypoplasia and severe EFE, the apex being formed by the right ventricle (B). In panel (C), the four-chamber view of the same fetus shows severe left ventricular hypoplasia in terms of a hypoplastic left heart syndrome making a univentricular palliation inevitable. EFE, endocardial fibroelastosis; LA, left atrium; LV, left ventricle; RA, right atrium; RV, right ventricle.