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. 2022 Jul 8;13:876235. doi: 10.3389/fphar.2022.876235

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Copyright © 2022 Zhou, Li, Shen, Gao, Zhuang, Zhang and Guo.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Storax inhibits caveolae-mediated transcytosis and attenuates the ultrastructural disruption of BBB at 6 h after stroke. (A) Representative images of the ultrastructure of basement membrane (BM), tight junctions (TJs), pericyte, astrocytic mitochondrial and caveolae in endothelial cells (ECs) under TEM observation at 6 h after stroke. (B–D) Quantification of the pericyte coverage, swelling rate of the astrocytic mitochondrial and the number of caveolae. Data were shown as the mean ± SEM. ^## p < 0.01, ^### p < 0.001 vs. Control group, *p < 0.05 vs. Storax group, n = 3.