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. 2022 Jul 2;13(8):1531–1546. doi: 10.1007/s13300-022-01285-1

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution-NonCommercial 4.0 International License, which permits any non-commercial use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by-nc/4.0/.

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Fig. 1 — Comparison of tissue (evaluated by SAF) and circulating (i.e., CML, pentosidine, MGH1) AGEs between patients with macroangiopathy and those without macroangiopathy. Data are presented in box plots comparing patients with macroangiopathy and those without macroangiopathy. Skin autofluorescence (SAF), which reflects the tissue content of advanced glycation end-products, is represented in box plot A and circulating AGE are represented in box plots B for pentosidine, C for carboxymethylation, and D for methylglyoxal, respectively