Table 1.

Summary table of the mechanisms resulting in oxidative stress in the different analyzed conditions.

Endometriosis	PCOS	Advanced Age	After Cancer Treatment
▪Macrophages recruited by apoptotic erythrocytes and endometrial cells are responsible for lipid peroxidation ▪Increased expression of nitric oxide synthase ▪Lower levels of peroxide dismutase and glutathione peroxidase in peritoneal fluid	▪Impaired matrix metalloproteinases activity ▪Abnormal ovarian extracellular matrix ▪Low levels of vitamin D	▪Loss of DNA repair ability ▪Decreased number of mitochondrial DNA copies ▪Upregulation of cell-free DNA levels	▪Radiotherapy: prolonged loss of granulosa cells, accelerated process of small vessel sclerosis and myointimal proliferation ▪Chemotherapy: DNA breaks, triggering apoptosis