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. 2022 May 24;39(8):1789–1802. doi: 10.1007/s11095-022-03295-x

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — Simulation results and clinical data describing the correspondence between the simulated histologic components of NAS with related outputs. The range of cytokeratin-cleaved K18 (cK18) and histologic ballooning (a), liver fat measured by MRI-PDFF and histologic steatosis (b), andCCL3 and histologic lobular inflammation scores. Red bars or symbols denote results from simulated patients while black symbols and black bars denote clinical data from Aida et al. (a), Middleton et al. (b), and du Plessis et al. (c).