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. Author manuscript; available in PMC: 2022 Jul 26.
Published in final edited form as: Med Hypotheses. 2013 Jul 24;81(4):532–535. doi: 10.1016/j.mehy.2013.06.026

Fig. 3.

Fig. 3.

N-formyl peptides from mitochondria (FMIT) and from bacteria trigger inflammation and vascular dysfunction via formyl peptide receptor (FPR) activation, contributing to the loss of control of vascular tone and to the development of sepsis.