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. Author manuscript; available in PMC: 2022 Nov 1.
Published in final edited form as: Gastroenterology. 2021 Jul 23;161(5):1513–1525. doi: 10.1053/j.gastro.2021.07.029

Figure 3: Graphs describing serum fatty acid ethyl ester concentrations during alcoholic pancreatitis.

Figure 3:

A: Change in FAEE levels over time from onset of pain. B: Box plots comparing FAEE concentrations in patients with elevated ethanol (Etoh.) levels (red dots, right side) to those in whom ethanol was not detectable (black dots, left side). P-value is computed from Wilcoxon rank sum test. C: Relationship between FAEEs and serum ethanol levels. D: Relationship between total FAEEs and NEFA in all patients with alcoholic pancreatitis including those with elevated alcohol (red dots). E-H: Correlations in patients who did not have elevated blood alcohol showing relationship between total non-esterified fatty acid (NEFA) to total FAEEs (E), individual FAEEs to NEFA (on the x-axis), which are F: Palmitic acid (PA) G: Stearic acid (SA) H: Oleic acid (OA). At the bottom right are shown the correlation coefficients, along with the p values. In A and C-H, the variable on the y-axis is treated as a dependent variable and the variable in the x-axis is treated as an independent variable. All the y-axes are plotted in logged scale with base 10. Dots are the actual data; blue lines are the slope from the linear regression, and gray band shows the 95% confidence interval. On the upper left are the slope (β) of the regression line and p value are shown on the left upper corner.