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. 2022 Jul 14;14(14):3431. doi: 10.3390/cancers14143431

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© 2022 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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Frequently investigated and affected genes within the subgroups MDS, AML, and. MDS/AML. Illustration of age, phenotype, type of RUNX1 germline variant and analyzed somatic variants for 53 HM cases with MDS and/or AML. For references to the individual patients please refer to the supplementary data. AML—acute myeloid leukemia, HM—hematologic malignancies, MDS—myelodysplastic syndrome, MDS/AML—patients who developed MDS and subsequently AML, NA—not available, RUNX1-FPD—familial platelet disorder with predisposition to hematologic malignancies. * some authors refer to the age of first reported symptoms and others to the age of genetic diagnosis.