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. 2022 Jul 25;13(8):1324. doi: 10.3390/genes13081324

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© 2022 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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AKBA-mediated neuroprotective effect on the amelioration of inflammatory cytokines in an experimental model of multiple sclerosis. Statistical values are represented in mean ± SEM (n = 6 each group). Statistical significant analysis was performed by posthoc Tukey’s test with one-way ANOVA. Here, * indicates versus vehicle control, sham control and AKBA100 perse group (p < 0.001); @ indicates versus EB group (p < 0.001); @# indicates versus EB + AKBA50 group (p < 0.001). (A): TNF-α level in brain homogenate; (B): TNF-α level in blood plasma; (C): IL-1β in brain homogenate; (D): IL-1β in blood plasma.

AKBA-mediated neuroprotective effect on the amelioration of inflammatory cytokines in an experimental model of multiple sclerosis. Statistical values are represented in mean ± SEM (n = 6 each group). Statistical significant analysis was performed by posthoc Tukey’s test with one-way ANOVA. Here, * indicates versus vehicle control, sham control and AKBA100 perse group (p < 0.001); @ indicates versus EB group (p < 0.001); @# indicates versus EB + AKBA50 group (p < 0.001). (A): TNF-α level in brain homogenate; (B): TNF-α level in blood plasma; (C): IL-1β in brain homogenate; (D): IL-1β in blood plasma.