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. 2022 Jun 21;4(1):vdac097. doi: 10.1093/noajnl/vdac097

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© The Author(s) 2022. Published by Oxford University Press, the Society for Neuro-Oncology and the European Association of Neuro-Oncology.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (https://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 2. — Survival analysis of IDH wild-type glioblastoma (GBM) cases according to PDGFRA and MGMTp status. (A) Survival analysis of patients with IDH wild-type GBM harboring either unmethylated or methylated MGMTp without PDGFRA gain/amplification. (B) Survival analysis of patients with IDH wild-type GBM harboring either unmethylated or methylated MGMTp with PDGFRA gain/amplification. (C) Kaplan-Meier analysis for OS of patients with and without PDGFRA gain/amplification in IDH wild-type GBMs with methylated MGMTp. (D) Kaplan-Meier survival analysis of patients with and without PDGFRA gain/amplification in IDH wild-type GBMs with unmethylated MGMTp.