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. 2022 May 26;11(4):1541–1557. doi: 10.1007/s40121-022-00654-4

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution-NonCommercial 4.0 International License, which permits any non-commercial use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by-nc/4.0/.

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Fig. 1 — Levels of bacterial translocation (a), inflammation (b) and cardiovascular risk markers (c) in the studied population compared with the control group, naïve group and ART-treated group. *p < 0.05 and ***p < 0.001 vs. control, ^##p < 0.01 and ^###p < 0.001 vs. ART. ART antiretroviral treatment, ICAM-1 intracellular adhesion molecule 1, IL-6 interleukin-6, LBP lipopolysaccharide binding protein, PAI-1 plasminogen activator protein 1, sCD14 soluble cluster of differentiation 14, TMAO trimethylamine N-oxide, TNF-alpha tumour necrosis factor alpha, VCAM-1 vascular cell adhesion molecule 1