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. 2022 Jul 30;20:341. doi: 10.1186/s12967-022-03540-2

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — The suppression of VEGFB gene inhibits AMPK phosphorylation level. A Venn diagram illustrates the number of loci with gained VEGFB and AMPK binding in liver. B Bioinformatics analysis of co-expressed genes of VEGFB and AMPK. C p-AMPK, AMPK, CaMKK2 in NAFLD mice model were analyzed by Western blot. D Protein expression level of p-AMPK/AMPK in NAFLD mice model. E Protein expression level of CaMKK2 in NAFLD mice model. F p-AMPK, AMPK, CaMKK2 in NAFLD cell model were analyzed by Western blot. G Protein expression level of p-AMPK/AMPK in NAFLD cell model. H Protein expression level of CaMKK2 in NAFLD cell model. N = 6 cases per group. D, E * indicates vs. SD, p < 0.05; ^# indicates vs. HFD, p < 0.05. G, H * indicates vs. NC, p < 0.05