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. 2022 May 10;12(8):jkac115. doi: 10.1093/g3journal/jkac115

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© The Author(s) 2022. Published by Oxford University Press on behalf of Genetics Society of America.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (https://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 2. — Gpdh1 is required for oocyte development and embryonic viability. a–c) Ovaries were dissected from 3-day old females and stained with DAPI. When compare to the Gpdh1^A10/+ control strain a), the ovaries of b) F1 generation Gpdh1^A10 mutants and c) Gpdh1^A10 M/Z mutants display b and c) fewer later stage oocytes and b) degenerating egg chambers (see arrow). d) The fecundity of F1 generation Gpdh1^A10 mutants and Gpdh1^A10 M/Z mutants is significantly decreased when compared with controls. e) Embryos produced by either F1 generation Gpdh1^A10 mutants or Gpdh1^A10 M/Z mutants exhibit significant mortality when compared with the controls and independent of paternal genotype. *P < 0.05. **P < 0.01. ***P < 0.001. P-values calculated using a Kruskal–Wallis tests followed by a Dunn’s test.