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. 2022 Aug 1;2(1):19. doi: 10.1186/s44149-022-00051-x

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig 4 — Cellular responses to TX98 and CA04 measured by IFN-γ ELISPOT assays. IFN-γ production by PBMC collected at 20 d.p.v. and 5 d.p.i. after incubation with UV-inactivated TX98 (a) or CA04 (b) virus particles. IFN-γ production by lung leukocytes isolated at 5 d.p.i. after incubation with UV-inactivated TX98 (c) or CA04 (d) virus particles. Results represent mean IFN-γ spots per 1x10⁶ live cells after subtracting spots counted in unstimulated wells. Differences between treatments were analyzed by Dunn’s multiple comparisons test. A statistically significant difference between two groups is indicated by different letters. Data are represented as mean ± SEM. Symbols represent individual pigs