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. 2022 Jul 18;12:921194. doi: 10.3389/fonc.2022.921194

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Copyright © 2022 Qi, Chen, Zhao, Chen, Niu and Shen

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Schematic diagram of representative ceRNA crosstalks function in GC (A), CRC (B) and EC (C). (A) ceRNA interaction regulates tumor cell proliferation, migration, invasion, apoptosis, or chemoresistance through PI3K/ATK, MAPK or Wnt/β-catenin signaling pathways, thereby exerting carcinogenic or tumor suppressor effects in GC. (B) ceRNA interaction contributes to CRC progression or chemoresistance by regulating autophagy process or pivotal pathways, such as Wnt/β-catenin, PI3K/ATK and JAK2/STAT3 signaling pathway. (C) ceRNA interaction promotes or inhibits EC progression by modulating cancer cell proliferation, migration, invasion, or apoptosis.