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. 2022 Jun 23;13(6):14057–14065. doi: 10.1080/21655979.2022.2062988

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© 2022 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 4. — ZNF503-AS1 overexpression promoted TGF-β1 expression. This figure shows the effects of high glucose environments on ZNF503-AS1 expression in ARPE-19 cells (a). ZNF503-AS1 overexpression was achieved in human retinal pigment epithelial ARPE-19 cells (b), upregulation of TGF-β1 and TGF-β receptor1 (TGFBR1) protein after ZNF503-AS1 overexpression (d) and ZNF503-AS1 expression in ARPE-19 cells treated with exogenous TGF-β1 (d).

Notes: *, p < 0.05; C, cells without transfection, NC, empty vector transfection; ZNF503-AS1, cells transfected with the ZNF503-AS1 expression vector.