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. 2015 Jan 9;23(4):679–691. doi: 10.1016/j.jfda.2014.07.011

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© 2015 Taiwan Food and Drug Administration

This is an open access article under the CC-BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig. 1 — (A) Effect of aspartame (40 mg/kg b.w) on superoxide dismutase (SOD) activity (units/mg of tissue protein) in rat liver. (B) Effect of aspartame (40 mg/kg b.w) on catalase (CAT) activity (H₂O₂ utilized/min/mg protein) in rat liver. (C) Effect of aspartame (40 mg/kg b.w) on reduced glutathione (GSH) concentration (μg of GSH/mg of protein) in rat liver. (D) Effect of aspartame (40 mg/kg b.w) on lipid peroxidation (LPO) (Moles of MDA/mg tissue) in rat liver. (E) Effect of aspartame (40 mg/kg b.w) on protein thiol (μg/mg protein) in rat liver. Comparison and analysis were done by the one-way analysis of variance, (n = 6) control group was compared with the MTX control group and aspartame MTX group. Significance fixed at p < 0.05. The data from various groups for the individual parameters are presented as bar diagram with mean ± SD. ^aAspartame treated group when compared to control significance. ^bMTX treated groups when compared to control significance. Control = MTX control–methotrexate treated group; MTX + Asp = methotrexate + aspartame treated group.