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. 2022 Jun 30;65(9):1483–1494. doi: 10.1007/s00125-022-05746-x

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — Estimates of the causal effects of genetically predicted type 2 diabetes and glycaemic traits on PCOS. The boxes denote the point estimates of the causal effects and the error bars denote the 95% CIs. The IVW approach was used in the primary analysis and the MR-Egger and weighted median approaches were used in sensitivity analyses. The ORs for PCOS were scaled to the per unit increase in log OR of type 2 diabetes and per unit increase in glycaemic traits. T2DM, type 2 diabetes mellitus