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. 2021 Sep 13;29(4):1702–1709. doi: 10.1007/s12350-021-02781-w

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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[¹⁸Fluor]fluordeoxyglucose ([¹⁸F]FDG) and five-year follow-up [¹⁸Fluor]SodiumFluoride ([¹⁸F]NaF) positron emission tomography/low dose computed tomography in a patient with type 2 diabetes mellitus. A Metabolic active cells are visualized by [¹⁸F]FDG uptake can be found in the liver, heart, and more focal in arterial tissue (pop-up). In arterial tissue, [¹⁸F]FDG uptake is a marker for inflammation which is considered an important factor in the development of CVD. B [¹⁸F]NaF uptake demonstrates skeletal activity and, next to that, this tracer also visualizes micro-calcification formation in the arterial wall (pop-up). Both nuclear tracers are excreted via the kidneys, ureters, and bladder. For image processing and analysis Affinity 2.0 was used (Hermes Medical Solutions)