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. 2022 Aug 2;12:307. doi: 10.1038/s41398-022-02039-0

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — a LPSD vs. frequency (averaged across all channels) for eyes closed (left) and open (right), both for the placebo and the active dose (mean ± SEM). b Same as in panel A, but binned using the following bands: delta (1–4 Hz), theta (4–8 Hz), alpha (8–12 Hz) and beta (12–20 Hz). c Global Lempel-Ziv complexity computed using the broadband EEG signal, compared between psilocybin and placebo conditions for eyes closed and open. d Topographic distribution of spectral power and Lempel-Ziv complexity for all combinations of active dose, placebo, eyes open and closed. e Results of the Local-Global ERP analysis. Left: global deviant minus local deviant ERPs located at AFz for the the placebo condition. Center: local deviant at AFz for placebo vs. psilocybin. Right: same as in panel C but for the global deviant. All ERP plots show mean ± SEM. The vertical dashed lines coincide with the timing of the last sound in the trial. *p < 0.05 for the comparison between placebo and active dose (corrected for multiple comparisons).