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. 2022 Aug 4;23:49. doi: 10.1186/s12868-022-00735-7

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 8 — rTMS stimulated MCM reduced infarct volumes and promoted functional recovery in MCAO mice, but weaken by let-7b-5p antagomir. Immediately after reperfusion and at 3 days post reperfusion, the MCAO mice received retroorbital intravenously injection of indicated MCM. At 7 days post reperfusion, ischemic regions were detected by TTC staining. a Representative image of TTC staining in the indicated groups. b Quantitative analysis of infarct volume. c The neurological deficit was evaluated by modified Neurological Severity Score (mNSS). n = 8 animals per group. Data are expressed as mean ± SD; **P < 0.01, ***P < 0.001, ns no significance