Skip to main content
. 2022 Jul 22;13:915575. doi: 10.3389/fendo.2022.915575

Figure 1.

Figure 1

Correlations between the circulating levels of AI, API, and the apolipoproteins by Spearman’s rank correlation coefficient analysis. (A): apoB/apoA-I (p < 0.001), apoC-II/apoC-III (p < 0.001), and apoA-II/apoA-I (p < 0.001) were positively correlated with AI. apoB/non–HDL-C (p < 0.001) and apoE/apoC-II were significantly negatively corelated with AI. (B): apoC-III (p < 0.001), apoE (p < 0.001), apoC-II, and apoB were positively correlated with AI. (C): apoB/apoA-I (p < 0.001), apoC-II/apoC-III (p < 0.001), and apoA-II/apoA-I (p < 0.001) were significantly positively correlated with API. apoB/non–HDL-C (p < 0.001) and apoE/apoC-II were significantly negatively correlated with API. (D): apoA1 (p < 0.001), apoC-III (p < 0.001), apoE (p < 0.001), apoC-II, and apoB were positively correlated with API, but apoA-I was significantly negatively correlated with API. The brown histograms on the right side represent the data distribution of AI and API. AI, atherogenic index was calculated as TC-(LDL-C)/HDL-C. API, atherogenic plasma index was defined as LDL-C/HDL-C. apo, apolipoprotein.