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. 2022 Aug 4;14:2323–2337. doi: 10.2147/CMAR.S363629

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© 2022 Zheng et al.

This work is published and licensed by Dove Medical Press Limited. The full terms of this license are available at https://www.dovepress.com/terms.php and incorporate the Creative Commons Attribution – Non Commercial (unported, v3.0) License (http://creativecommons.org/licenses/by-nc/3.0/). By accessing the work you hereby accept the Terms. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed. For permission for commercial use of this work, please see paragraphs 4.2 and 5 of our Terms (https://www.dovepress.com/terms.php).

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Overexpressed tiRNA-Val-CAC-001 inhibited gastric cancer (GC) cells metastasis, proliferation and apoptosis. (A) Transwell invasion assay indicated overexpressed tiRNA-Val-CAC-001 inhibited invasion of GC cells (*P<0.05). (B) Transwell migration assay indicated overexpressed tiRNA-Val-CAC-001 inhibited migration of GC cells (*P<0.05). (C) Colony formation assay indicated overexpressed tiRNA-Val-CAC-001 inhibited proliferation of GC cells (*P<0.05). (D) Overexpressed tiRNA-Val-CAC-001 promoted apoptosis of GC cells. (E) CCK-8 assay was used to examine cell proliferation in GC cell lines transfected with tiRNA-Val-CAC-001 mimics or mimics-NC (*P<0.05).

Abbreviations: mimics, tiRNA-Val-CAC-001 mimics; mimics-NC, negative control of mimics.