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. 2022 Aug 8;12:13550. doi: 10.1038/s41598-022-17767-y

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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The effects of wBCC-EV on BCC, EC, and EPC. (A) The cell migration of BCC treated with BCC-EV or wBCC-EV, n = 3, *p < 0.05, ***p < 0.001. (B) The cell proliferation of BCC treated with BCC-EV or wBCC-EV for 24 h, n = 3, ***p < 0.001. (C) The sphere formation of BCC treated with BCC-EV or wBCC-EV for 24 h, n = 3, ***p < 0.001. (D) The tumor metastatic ability of the BCC treated with BCC-EV or wBCC-EV in vivo, n = 4, *p < 0.05, **p < 0.01, ***p < 0.001. (E) The tube formation of EC treated with BCC-EV or wBCC-EV, n = 9, **p < 0.01. (F) The angiogenic gene expression of EC treated with BCC-EV or wBCC-EV, n = 3, *p < 0.05. (G) The tube formation of EPC treated with BCC-EV or wBCC-EV, n = 9, **p < 0.01. (H) The cell migration of EPC treated with BCC-EV and wBCC-EV, n = 3, ***p < 0.001.