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. 2022 Aug 8;19:202. doi: 10.1186/s12974-022-02560-5

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — ASK1 cKO altered the mRNA expression of pro- and anti-inflammatory markers in the hippocampus(3d). A The mRNA levels of pro-inflammatory Mi/Mϕ specific marker (CD16, CD32). B The mRNA levels of released pro-inflammatory cytokines (TNFa, IL-1β and IL-6). C The mRNA levels of anti-inflammatory Mi/Mϕ specific markers (Arg-1, Ym1/2). D The mRNA levels of released anti-inflammatory cytokines (IL-13, TGFβ) and chemokines (CCL22). All data are expressed as the mean ± SD. ***p < 0.001, **p < 0.01, and *p < 0.05, ns no significance, as indicted. n = 4 in the WT sham or cKO sham groups, n = 7 in the WT KA group, n = 6 in the cKO KA group. p values are based on the one way ANOVA and Kruskal–Wallis ANOVA(A-D)