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. 2022 May 20;32(4):312–319. doi: 10.4103/ijn.ijn_234_21

Table 2.

Pathological characteristics and treatment

Pretransplant biopsy (n=41)
 HAAS staging
  IV 38.8% (n=7)
  1V 61.1% (n=34)
 MEST score
  M1 33.3% (n=6)
  E1 5.5% (n=1)
  S1 50% (n=9)
  T1 44.4% (n=8)
  T2 11.1% (n=2)
  C1 22.2% (n=4)
  C2 11.1% (n=2)
0%
Posttransplant biopsy (n=51)
 HAAS staging
  I 17.6% (n=9)
  II 11.8% (n=6)
  III 25.5% (n=18)
  IV 35.3% (n=18)
  V 9.8% (n=5)
 MEST score
  M1 41.2% (n=21)
  E1 5.9 (n=3)
  S1 49% (n=25)
  T1 17.6% (n=9)
  T2 0% (n=0)
  C1 3.9% (n=2)
  C2 0% (n=0)
 Proteinuria range
  <1 g 15.7% (n=8)
  1-3 g 43.1% (n=22)
  >3 g 41.2% (n=21)
 Treatment
  ACE/ARB 100% (n=51)
  Omega-3 fatty acids 21.6% (n=11)
  High-dose oral steroids 5.9% (n=3)
  IV MPS 3.9% (n=2)

HAAS =, MEST=mesangial and endocapillary hypercellularity, segmental sclerosis, and interstitial fibrosis/tubular atrophy, ACE=angiotensin-converting enzyme, ARB=angiotensin II receptor blocker, MPS=mucopolysaccharidosis