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. 2022 Aug 10;608(7922):360–367. doi: 10.1038/s41586-022-05023-2

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Extended Data Fig. 8 — a, Differentially expressed genes from benign clone A and altered clone C. Using a two-sided Wilcoxon Rank-Sum test. b, Top 10 pathways identified by geneset enrichment analyses (GSEA) from clone A vs clone C. c, Top ranked enrichment pathway from GSEA. d, Differentially expressed genes from benign clone A and tumour clones E, F and G. Using a two-sided Wilcoxon Rank-Sum test. e, Top 10 pathways identified by GSEA from clone A vs clones E, F and G. f, Top ranked enrichment pathway from GSEA. g, Venn-Diagram of genes from differential gene expression analyses identified only in benign clone A vs altered benign clone C analysis (left), benign clone A vs tumour clones E, F and G (right).