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. 2022 Jul 29;9:954990. doi: 10.3389/fmed.2022.954990

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Copyright © 2022 Gandhi, Shrestha Palikhe and Vliagoftis.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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PAR-2 activated airway epithelial cells release a variety of inflammatory mediators that may induce airway inflammation, airway hyperresponsiveness and airway remodeling. The numbers in brackets after the name of specific inflammatory mediators indicate the reference of the main text where that mediator is discussed. AHR, airway hyperresponsiveness; CCL2, CC motif chemokine ligand 2; DC, dendritic cell; GM-CSF, granulocyte-macrophage colony-stimulating factor; ILC2, type 2 innate lymphoid cells; IL-25, interleukin 25; IL-8, interleukin 8; LTC4, leukotriene C4; MMP-9, matrix metalloproteinase 9; TSLP, thymic stromal lymphopoietin.