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. 2022 Aug 12;19:204. doi: 10.1186/s12974-022-02566-z

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 10 — Factors secreted by CT26 cells and inflammatory changes in CT26 tumor-bearing mice. The change in different inflammatory mediators was measured by proteome profiler array and presented as fold-change in signal intensity compared to mean control values (n = 3/group, in duplicates). a Inflammatory mediators found to be secreted by CT26 cells (factors with elevated signal intensity in conditioned media vs un-conditioned media, as presented by fold-increase). b Increased factors in blood samples collected 3 weeks after CT26 inoculation. Arrows mark mediators detected in CT26-injected mouse serum that were also detected in CT26-conditioned media (a)