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. 2022 Jul 25;11(15):e026426. doi: 10.1161/JAHA.122.026426

Figure 4. Effect of fludrocortisone in mice deficient for hepatic AGT (angiotensinogen).

Figure 4

A, AGT protein abundance in the liver, (B) plasma AGT concentrations, (C) AGT protein abundance in epididymal white adipose tissue (eWAT), (D) systolic blood pressure (SBP), and (E) representative immunoblots of mice deficient for liver angiotensinogen (hepAGT−/−) and floxed mice not expressing Cre (hepAGT+/+) that were administered with either vehicle or fludrocortisone as indicated. Data (mean±SEM of 7–14 per group) were analyzed using a 1‐way ANOVA and post hoc Bonferroni (**P≤0.01, ***P≤0.001, ****P≤0.0001). NS indicates not significant.