Table 3.

Autophagy-related (AR) signatures of lncRNAs proposed as biomarkers in HNSCC.

AR LNCRNAS	INFLUENCE	REFERENCE
PTCSC2, AC099850.3, LINC01963, RTCA-AS1, AP002884.1, UBAC2-AS1, AL512274.1, MIR600HG, AL354733.3	The overall survival of the high-risk group was significantly lower than that of the low-risk group. The signature-based on autophagy/related lncRNAs potentially acts as an independent prognostic indicator for patients with OSCC/OPSCC.	(13)
ATG12, BACN1, MAP1LC3B	All three autophagy-related lncRNAs have prognostic value with respect to HNSCC, and their related pathways may be involved in regulating HNSCC prognosis.	(191)
MIR4435-2HG, PCED1B-AS1, AL512274-1, MYOSLID, LINC01871, LINC02541, AC012236-1, C5orf66-AS1, AC004687-1, AL354836.1, LINC02454, AC024075.2, LINC00460, AATBC, ITGB2-AS1, MIR9-3HG, AF131215.5	Differentially expressed genes (DEGs) between high- and low-risk groups were mainly enriched in immune-related pathways and regulated by a PAF-lncRNA-directed ceRNA network.	(12)
AC008115.3, AL139158.2, AC136475.2, AL160006.1, AL357033.4, AC007991.2, AC104083.1, AL139287.1, AL450992.2, LINC00958, AC103702.2, PSMA3-AS1, UBAC2-AS1	Overall survival in the high-risk group was shorter than the low-risk group. Gene set enrichment analysis (GSEA) and gene ontology (GO) functional annotation proved that autophagy-related pathways are mainly enriched in the high-risk group.	(140)