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. 2022 Aug 3;12:967000. doi: 10.3389/fonc.2022.967000

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Copyright © 2022 Li, Chen, Zhou, Maimaitirexiati, Yan, Li, Maimaitiyimin, Zhou, Ren, Liu, Mainike, Zhou and Ding

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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ZEB2 is a downstream target of miR-26a-5p in osteosarcoma cells. (A): The potential binding sites between miR-26a-5p and ZEB2. (B): Left panel: RT-PCR data showed that miR-26a-5p mimics attenuated the mRNA levels of ZEB2 in MG63 cells. miR-26a-5p inhibitors elevated ZEB2 mRNA levels in U2OS cells. Right panel: RT-PCR data showed that SCAMP1-overexpressing cells had the high expression of ZEB2 in MG63 cells. In shR-SCAMP1 transfected U2OS cells, ZEB2 mRNA levels were remarkable downregulated. **p<0.01 vs. control group.