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. 2022 Aug 3;13:958273. doi: 10.3389/fimmu.2022.958273

Table 3.

Overview of publications in the disease clusters Vascular diseases, Pregnancy, Cancer and Various.

Disease entity oc Disease model Gene manipulation and genetic background Sex Ref
Vascular diseases
Atherosclerosis - atherosclerosis C1q-/-/Ldlr-/- f (125)
- atherosclerosis C1q.Ldlr-/- , C1q.sIgM.Ldlr-/- f (126)
- woundhealing f/m (127)
- ALI f (128)
- primary hemostasis f/m (129)
Pregnancy
Fetal loss - fetal loss f (130)
+ fetal loss C1q/fD -/- f (131)
= fetal loss f (132)
PE - PE f (133)
- PE f (134)
- PE f (135)
Cancer
Solid tumor + melanoma f/m (136)
= breast cancer neuT+-C1q-/-, BALB/c f (137)
+ ccRCC f/m (138)
Tumor therapy - immunotherapy f/m (139)
= immunotherapy f/m (140)
= immunotherapy m (141)
- immunotherapy SCID/C1q-/- f (142)
Various
Skin = burn injury f/m (143)
+ epidermolysis bullosa BALB/c f/m (144)
+ muscle regeneration f/m (145)
Pulmo - COPD f (146)
+ pulmonary fibrosis f/m (147)
= AA amyloidosis C57B L 6×129/SV f/m (148)
= adipose inflammation f (149)

Each cluster is subdivided according to disease and organ manifestation, respectively. Disease outcome (oc) of C1qKO mice compared to wt and/or C1q sufficient mice in the investigated disease model is given as “+” respectively turquoise =beneficial, “-” respectively ocher =detrimental, “=“ respectively grey=no effect. The overall outcome on the disease entity is similarly color coded using lighter shades for ambiguous group results. Genetic modifications other than C1qKO and genetic background other than C57BL/6 are listed explicitly. In studies with several C1q deficient mice, all C1q deficient mice are listed. Sex as indicated in the study (f=female only, m=male only, f/m=mixed gender); if not mentioned explicitly by the study, mixed gender was assumed. ALI, acute lung injury; AA, amyloid A protein; COPD, chronic obstructive pulmonary disease; PE, preeclampsia.