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. 2022 Aug 17;13(8):715. doi: 10.1038/s41419-022-05132-w

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 8 — A NDUFA4 expression in AGS cells treated with 100 μM 3-Deazaadenosine (DAA) for 0, 12, 24, and 48 h. B–F AGS cells were transfected with METTL14 siRNA. The whole m6A level (B), m6A level of NDUFA4 3’UTR (C), and abundance of NDUFA4 3’UTR (D) were detected. The expression of METTL3 (E) and NDUFA4 (F) was detected. G–H IGF2BP1 expression (G) and NDUFA4 mRNA stability (H) in AGS cells transfected with IGF2BP1 shRNA. I The interaction between IGF2BP1 and NDUFA4 3′UTR was detected by RIP assay. ***P < 0.001 vs 0 h, siNC, shNC or IgG.