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. 2022 Aug 4;13:969499. doi: 10.3389/fmicb.2022.969499

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Copyright © 2022 Wei, Shu, Sun, Chen, Li, Luo, Yang and Tan.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Deleting Bclae1 changes the growth, morphology, and reproductive development. (A) The TB-31, ΔBclae1, ΔBclae1-C (X7), and Bclae1-OE-X4 mutants were photographed after 6 days of PDA growth under DD culture conditions, and the colony diameters were calculated (B). Shown are means and SEM, n = 3 independent biological replicates. ^*p < 0.05 versus the colony diameter of the TB-31 group. (C) Under DD or LD conditions, TB-31, ΔBclae1, ΔBclae1-C (X7), and Bclae1-OE-X4 mutants were counted for sclerotia and conidia numbers after 14 days of PDA growth. Shown are means and SEM, n = 3 independent biological replicates. ^*p < 0.05 versus the TB-31 group. (D) RT-qPCR examining the transcriptional levels of Bccry2, BcPKS13, and Bcltf2 in TB-31 and ΔBclae1. Shown are means and SEM, n = 3 independent biological replicates. ^**p < 0.01/^***p < 0.001 versus the same genes of the TB-31 group.