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. 2022 Aug 18;21:75. doi: 10.1186/s12944-022-01684-0

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Imiquimod-mediated skin inflammation in a psoriasis mouse model. Back skin shaved in CETP-Tg, PLTP-Tg, and WT mice was exposed to IMQ cream every day. A–C Phenotypes of the back skin of mice following treatment for five days consecutively. D–E Back skin clinical cumulative score (TLPSS; erythema + scaling + thickness). Symbols represent the mean ± SD; n = 6/group; *P < 0.05 vs. WT group; IMQ, imquimod; CETP, cholesteryl ester transfer protein; PLTP, phospholipid transfer protein; TLPSS, target lesion psoriasis severity score; WT, wild-type; Tg, transgenic