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. 2022 May 5;122(8):1314–1325. doi: 10.1055/a-1759-9962

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The Author(s). This is an open access article published by Thieme under the terms of the Creative Commons Attribution-NonDerivative-NonCommercial License, permitting copying and reproduction so long as the original work is given appropriate credit. Contents may not be used for commercial purposes, or adapted, remixed, transformed or built upon. ( https://creativecommons.org/licenses/by-nc-nd/4.0/ )

This is an open-access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivatives License, which permits unrestricted reproduction and distribution, for non-commercial purposes only; and use and reproduction, but not distribution, of adapted material for non-commercial purposes only, provided the original work is properly cited.

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Fig. 6. — Local inflammation 24 hours after experimental ICH. Neutrophil infiltration in the hemorrhage area of (A) nonanticoagulated, (B) warfarin, and (C) rivaroxaban anticoagulated mice. (D) Neutrophil infiltration in the hemorrhage area of wild-type ( WT ) and MMP-10-deficient ( Mmp10 −/− ) animals. (E) Representative immunofluorescence images showing neutrophils ( red ) and DAPI ( blue ) in the hemorrhage area ( white dots ) of WT and Mmp10 −/− mice. Scale = 200 µm. Magnification images of selected areas (*). Scale = 20 µm. Mean ± SD, * p < 0.05 vs. saline and ^## p < 0.01 vs. WT using Kruskal–Wallis and Mann–Whitney U-test, n ≥ 4/group. ICH, intracranial hemorrhage; SD, standard deviation.