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. 2022 Aug 8;12:922332. doi: 10.3389/fonc.2022.922332

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Copyright © 2022 Hu, Wang, Ma, Zhang and Xue

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Preliminary screening of cuproptosis-related genes indicative of LUAD survival. (A) Kaplan-Meier curves for OS of two LUAD subgroups automatically clustered by transcriptomic profiles. (B) Enrichment analysis showing characteristics of two LUAD subgroups. Cluster B with poorer five year-survival are enriched in pathways including DNA repair, MYC targets, MTORC1 signaling, E2F targets, and G2M checkpoints. (C) Expression of six cuproptosis biomarkers related to poor overall survival and their relationships with clinicopathological features. (D) GO enrichment analysis of the six-cuproptosis marker genes.