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. 2022 Aug 9;9:925816. doi: 10.3389/fcvm.2022.925816

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Copyright © 2022 Bhatnagar, Dixit, Yang and Sallam.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Host factors such as physiology and lifestyle, tumor factors that promote lipid dysregulation, and nuances of cancer therapeutics likely influence dyslipidemia in cancer. Dyslipidemia in cancer is likely due to host physiology and lifestyle, tumor processes that promote lipid dysregulation, and effects of cancer therapeutics on key points of lipid metabolism. CHIP, Clonal hematopoiesis of indeterminate potential; LXR-alpha, Liver X receptor alpha; PPAR-gamma, Peroxisome-proliferator activated receptor gamma; LDL, Low density lipoprotein; HMGCR, 3-hydroxy-3-methylglutaryl coenzyme A reductase; ABCA-1, Adenosine triphosphate binding cassette subfamily A member 1; Created using Biorender.com.