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. 2022 May 12;49(11):3627–3638. doi: 10.1007/s00259-022-05821-w

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© The Author(s) 2022, corrected publication 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Cell killing efficacy of [²²⁵Ac]Ac-PSMA-I&T and [¹⁷⁷Lu]Lu-PSMA-I&T. A Clonogenic survival of cells treated with increasing concentrations of [²²⁵Ac]Ac-PSMA-I&T for 3h. B Clonogenic survival of non-treated cells or cells treated with 0.37 kBq/mL [²²⁵Ac]Ac-PSMA-I&T, with or without ×1000 excess of unlabeled PSMA-I&T (block). C Clonogenic survival of cells treated with increasing concentrations of [¹⁷⁷Lu]Lu-PSMA-I&T for 3h. D Clonogenic survival of non-treated cells or cells treated with 0.4 MBq/mL [¹⁷⁷Lu]Lu-PSMA-I&T, with or without ×1000 excess of unlabeled PSMA-I&T (block). For all graphs, experiments were repeated three times. Error bars represent standard error of the mean (SEM). Asterisks indicate significance compared to non-treated (**** p ≤ 0.0001)