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. 2022 Aug 24;14:115. doi: 10.1186/s13195-022-01058-9

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Plasma apoE levels per diagnostic group. Plasma apoE levels in controls, MCI-MCI, MCI-ADD and ADD patients (a) and in groups based on the A/T/N classification (b) and the Aβ_1-42 status (c). Data is presented as median (minimum–maximum). Group differences were assessed using ANOVA (Tukey HSD post hoc) (a), the Kruskal–Wallis test followed by the Dunn’s test uncorrected/corrected for multiple comparisons using Bonferroni correction (b), or Mann–Whitney U test (c). Star marked p-values obtained after accounting for the APOE genotype of the studied subjects. The A-/T-/N + and A + /T + /N- groups were excluded from the statistical analysis due to low n-numbers (n = 2, in each group)