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. 2022 Aug 12;13:953206. doi: 10.3389/fphys.2022.953206

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Copyright © 2022 Sharawy, Imam, Aboulhoda, Khalifa, Morcos, Abd Algaleel, Moustafa, Abdelbaset and Shoukry.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Sepsis-induced encephalopathy enhanced microgliosis and neuronal degeneration. H&E-stained sections of the different brain regions showing darkly-degenerated, apoptotic neurons (arrows) and glial cell proliferation (circles). The dentate gyrus shows marked microgliosis (g). The insets in the cerebral cortex show a picture of subependymal microgliosis in the CLP 24 h groups, and foci of focal cortical microgliosis in the CLP 72 h groups. Meningeal congestion (M) is also observed in the cerebral cortex of the CLP 24 h group. The blood capillaries (C) exhibit interruption of their endothelial lining with widening of Virchow Robin’s (perivascular) space.