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. 2022 Aug 25;23(Suppl 6):616. doi: 10.1186/s12864-022-08828-7

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — UBCS statistic for human chromosome 2 and Great Ape genomes. (Top panel) UBCS statistic for substitutions derived in human are depicted as dots. Lines are the UBSC statistic values smoothed using loess regression. HSA2 shows the peak of the UBCS values near the ancestral fusion site. Atypical central peak occurs for the UBCS statistic computed using comparisons of the human to all Great Ape genomes. (Bottom panel) Values of $\hat{p}$ parameter (proportion of weak-to-strong in all substitutions) for every 1 Mb window of substitution derived in human chromosome 2