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. 2022 Aug 29;8:70. doi: 10.1038/s41522-022-00332-w

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — a Cytotoxicity of Lynronne 1, 2 and P15s against human cell lung cells IMR90 (human bronchial fibroblasts). b Cytotoxicity of Lynronne 1, 2 and P15s against BEAS-2B (human lung epithelial cells). In all cases, values are from three independent replicates; results are expressed as means ± standard deviation. c AMP membrane permeabilisation activity at 4× MIC against PAO1 measured by propidium iodide method over time (d) AMP membrane permeabilisation activity at 4× MIC against LES431 measured by propidium iodide method over time. In both cases, values are from three independent replicates; results are expressed as means ± standard deviation (when not visible, this is due to lack of variability).