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. 2022 Aug 29;13(8):746. doi: 10.1038/s41419-022-04956-w

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Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — A The circFKBP5 with and without miR-513a-5p binding sites (CircNet). B qRT-PCR was performed to show quantification of circFKBP5 after oe-mutant circFKBP5 in EnzS1-C4-2 (left panel) and EnzR1-C4-2 (right panel) cells. C WB showed that, compared to wild type, oe-mutant circFKBP5 failed to increase PD-L1 expression significantly in CRPC cells. *p < 0.05, **p < 0.01.