Table 3.
Sequelaes detected in children with congenital toxoplasmosis attended at Outpatient Clinic Hospital of the State University of Londrina, Londrina, Paraná, from January 2000 to December 2010.
| Sequelae | Children (n = 29) |
|
|---|---|---|
| n | % | |
| Not detectablea | 9 | 31.1 |
| Detectable | 20 | 68.9 |
| Visual | 16 | 55.2 |
| Delay of psychomotor development | 9 | 31.0 |
| Convulsionb | 8 | 27.5 |
| Motor dysfunction | 4 | 13.8 |
| Hyperactivity and/or deficit of attention | 3 | 10.3 |
| Precocious puberty | 2 | 6.9 |
| Hypothyroidism | 1 | 3.4 |
| Ventricular peritoneal shunt | 1 | 3.4 |
| Hearing damagec | 3 | 50.0 |
a
Two children lost the follow-up.
b
Four children presented convulsion during the first month of life.
c
Detected in six children, all of them with hearing damage also presented concomitant neurological sequelae; 1/31 (3.4%) child co-infected with HIV-1 who died with 12 months of life due to herpetic encephalitis and severe sepsis.