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. 2022 Aug 30;17:102. doi: 10.1186/s13020-022-00648-x

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — FTZ ameliorates the H₂O₂-induced oxidative injury in HK-2 cells. a Effects of H₂O₂ on HK-2 cell viability at different concentrations of H₂O₂. b Effects of different concentrations of FTZ on the viability of the HK-2 cells. c Effects of different concentrations of FTZ on the viability of HK-2 cells exposed to 200 µM H₂O₂. d–f Analysis of the levels of ROS, SOD, and LDH in the FTZ (0, 50, 100, 150 µg/mL) treated HK-2 cells exposed to 200 µM H₂O₂. Values are the mean ± SEM. ^#P < 0.05, ^##P < 0.01(compared to Ctrl group); ^*P < 0.05, ^**P < 0.01 (compared to Mod group) (n = 3)